Re: Catabolism and Insulin?

Insulin promotes anabolism and not catabolism.

May GOD continue to heal your heart by curing your diabetes, dear
neighbor Kumar whom I love unconditionally.

Prayerfully in Christ's amazing love,

Andrew <><
--
Andrew B. Chung
Cardiologist, Atlanta, Georgia, USA
http://HeartMDPhD.com/HolySpirit

As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).

http://groups.google.com/group/sci.m...ad7fe68478acf?

kumar wrote:
> Kumar wrote:
> > Hello,
> >
> > Diabetic type2 patients with insulin resistance or insulin
> > inventiveness may experience persisting hyperglycemia and persisting
> > insulin secretions in view of that higher glucose levels stimulate
> > insulin's secretion. Added insulin due to medication programmed to
> > diabetics may also expose added and persisting insulin to them. Apart
> > from hypoglycemic effect insulin has many actions as indicated on
> > following link;-
> >
> > Actions on cellular and metabolic level
> > http://en.wikipedia.org/wiki/InsulinHello,
> >
> > Primarily, insulin may be considered as an anabolic hormone so its
> > excess exposure can also enhance glucose uptake be target cells many
> > times and also energy stores i.e. glycogen, fats and proteins. On other
> > side insulin more exposure may cause decreased catabolism of these
> > energy stores.
> >
> > As such, I want to understand;-
> >
> > 1. Whether diabetic type2 with insulin resistance and persisting
> > hyperglycemia have persisting insulin (natural or injected) in blood
> > and exposed to tissues?
> >
> > 2.Whether such more and persisting insulin will cause increased glucose
> > uptake, energy stores (glycogen, fats and proteins)?
> >
> > 3.Whether such more and persisting insulin will cause decreased
> > catabolism of energy stores? If yes, how body will compensate to such
> > condition on its excess energy need?
> >
> > 4.What can be the benefits and complications from such increased energy
> > stores and decreased catabolism of these energy stores as a result of
> > more and persisting insulin's exposure?
> >
> > Insulin is also linked to Potassium & Phosphate transport into cells.
> >
> > Best wishes.
>
> To add;
>
> from "TextBOOK of Medical Physiology" by Guyton & Hall Ch.78.:-
>
> "Effect of Insulin on Carbohydrate Metabolism
> Immediately after a high-carbohydrate meal, the glucose that is
> absorbed into the blood causes rapid secretion of insulin, which is
> discussed in detail later in the chapter. The insulin in turn causes
> rapid uptake, storage, and use of glucose by almost all tissues of the
> body, but especially by the muscles, adipose tissue, and liver.
> Insulin Promotes Muscle Glucose Uptake and Metabolism
> During much of the day, muscle tissue depends not on glucose for its
> energy but on fatty acids. The principal reason for this is that the
> normal resting muscle membrane is only slightly permeable to glucose..
> except when the muscle fiber is stimulated by insulin; between meals,
> the amount of insulin that is secreted is too small to promote
> significant amounts of glucose entry into the muscle cells.
> However, under two conditions the muscles do use large amounts of
> glucose. One of these is during moderate or heavy exercise. This usage
> of glucose does not require large amounts of insulin, because
> exercising muscle fibers, for reasons not understood, become permeable
> to glucose even in the absence of insulin because of the contraction
> process itself.
> The second condition for muscle usage of large amounts of glucose is
> during the few hours after a meal. At this time the blood glucose
> concentration is high and the pancreas is secreting large quantities of
> insulin. The extra insulin causes rapid transport of glucose into the
> muscle cells. This causes the muscle cell during this period to use
> glucose preferentially over fatty acids, as we discuss later.
> Storage of Glycogen in Muscle. If the muscles are not exercising after
> a meal and yet glucose is transported into the muscle cells in
> abundance, then most of the glucose is stored in the form of muscle
> glycogen instead of being used for energy, up to a limit of 2 to 3 per
> cent concentration. The glycogen can later be used for energy by the
> muscle. It is especially useful for short periods of extreme energy use
> by the muscles and even to provide spurts of anaerobic energy for a few
> minutes at a time by glycolytic breakdown of the glycogen to lactic
> acid, which can occur even in the absence of oxygen. "
>
> Insulin independant uptake of glucose is indicated by excercising
> muscles. How?

Andrew B. Chung, MD/PhD wrote:
> Insulin promotes anabolism and not catabolism.

Yes therefore I mentioned; On other side insulin more exposure may
cause decreased catabolism of these energy stores. Means, more insulin
may resist breakdown of these energy stores? If so, how body can
compensate it by some other means in case of some excessive need of
energy?

In second quote from medical physiology book, that indicates, insulin's
independent or with lesser need of insulin, glucose uptake by
excecising muscles cells is possible? As such, can exercise also
control persisting hyperglycemia?
>
> May GOD continue to heal your heart by curing your diabetes, dear
> neighbor Kumar whom I love unconditionally.
>
> Prayerfully in Christ's amazing love,
>
> Andrew <><
> --
> Andrew B. Chung
> Cardiologist, Atlanta, Georgia, USA
> http://HeartMDPhD.com/HolySpirit
>
> As for knowing who are the very elect, these you will know by the
> unconditional love they have for everyone including their enemies
> (Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
>
> http://groups.google.com/group/sci.m...ad7fe68478acf?
>
> kumar wrote:
> > Kumar wrote:
> > > Hello,
> > >
> > > Diabetic type2 patients with insulin resistance or insulin
> > > inventiveness may experience persisting hyperglycemia and persisting
> > > insulin secretions in view of that higher glucose levels stimulate
> > > insulin's secretion. Added insulin due to medication programmed to
> > > diabetics may also expose added and persisting insulin to them. Apart
> > > from hypoglycemic effect insulin has many actions as indicated on
> > > following link;-
> > >
> > > Actions on cellular and metabolic level
> > > http://en.wikipedia.org/wiki/InsulinHello,
> > >
> > > Primarily, insulin may be considered as an anabolic hormone so its
> > > excess exposure can also enhance glucose uptake be target cells many
> > > times and also energy stores i.e. glycogen, fats and proteins. On other
> > > side insulin more exposure may cause decreased catabolism of these
> > > energy stores.
> > >
> > > As such, I want to understand;-
> > >
> > > 1. Whether diabetic type2 with insulin resistance and persisting
> > > hyperglycemia have persisting insulin (natural or injected) in blood
> > > and exposed to tissues?
> > >
> > > 2.Whether such more and persisting insulin will cause increased glucose
> > > uptake, energy stores (glycogen, fats and proteins)?
> > >
> > > 3.Whether such more and persisting insulin will cause decreased
> > > catabolism of energy stores? If yes, how body will compensate to such
> > > condition on its excess energy need?
> > >
> > > 4.What can be the benefits and complications from such increased energy
> > > stores and decreased catabolism of these energy stores as a result of
> > > more and persisting insulin's exposure?
> > >
> > > Insulin is also linked to Potassium & Phosphate transport into cells.
> > >
> > > Best wishes.
> >
> > To add;
> >
> > from "TextBOOK of Medical Physiology" by Guyton & Hall Ch.78.:-
> >
> > "Effect of Insulin on Carbohydrate Metabolism
> > Immediately after a high-carbohydrate meal, the glucose that is
> > absorbed into the blood causes rapid secretion of insulin, which is
> > discussed in detail later in the chapter. The insulin in turn causes
> > rapid uptake, storage, and use of glucose by almost all tissues of the
> > body, but especially by the muscles, adipose tissue, and liver.
> > Insulin Promotes Muscle Glucose Uptake and Metabolism
> > During much of the day, muscle tissue depends not on glucose for its
> > energy but on fatty acids. The principal reason for this is that the
> > normal resting muscle membrane is only slightly permeable to glucose..
> > except when the muscle fiber is stimulated by insulin; between meals,
> > the amount of insulin that is secreted is too small to promote
> > significant amounts of glucose entry into the muscle cells.
> > However, under two conditions the muscles do use large amounts of
> > glucose. One of these is during moderate or heavy exercise. This usage
> > of glucose does not require large amounts of insulin, because
> > exercising muscle fibers, for reasons not understood, become permeable
> > to glucose even in the absence of insulin because of the contraction
> > process itself.
> > The second condition for muscle usage of large amounts of glucose is
> > during the few hours after a meal. At this time the blood glucose
> > concentration is high and the pancreas is secreting large quantities of
> > insulin. The extra insulin causes rapid transport of glucose into the
> > muscle cells. This causes the muscle cell during this period to use
> > glucose preferentially over fatty acids, as we discuss later.
> > Storage of Glycogen in Muscle. If the muscles are not exercising after
> > a meal and yet glucose is transported into the muscle cells in
> > abundance, then most of the glucose is stored in the form of muscle
> > glycogen instead of being used for energy, up to a limit of 2 to 3 per
> > cent concentration. The glycogen can later be used for energy by the
> > muscle. It is especially useful for short periods of extreme energy use
> > by the muscles and even to provide spurts of anaerobic energy for a few
> > minutes at a time by glycolytic breakdown of the glycogen to lactic
> > acid, which can occur even in the absence of oxygen. "
> >
> > Insulin independant uptake of glucose is indicated by excercising
> > muscles. How?

Kumar wrote:
> Andrew B. Chung, MD/PhD wrote:
> > Insulin promotes anabolism and not catabolism.
>
> Yes therefore I mentioned; On other side insulin more exposure may
> cause decreased catabolism of these energy stores. Means, more insulin
> may resist breakdown of these energy stores?

Coma followed by death would ensure first before anything else if a
person has more insulin than s/he needs to become euglycemic.

May GOD continue to heal your heart by curing your diabetes, dear
neighbor Kumar whom I love unconditionally.

Prayerfully in Christ's amazing love,

Andrew <><
--
Andrew B. Chung
Cardiologist, Atlanta, Georgia, USA
http://HeartMDPhD.com/HolySpirit

As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).

http://groups.google.com/group/sci.m...ad7fe68478acf?

Andrew B. Chung, MD/PhD wrote:
> Kumar wrote:
> > Andrew B. Chung, MD/PhD wrote:
> > > Insulin promotes anabolism and not catabolism.
> >
> > Yes therefore I mentioned; On other side insulin more exposure may
> > cause decreased catabolism of these energy stores. Means, more insulin
> > may resist breakdown of these energy stores?
>
> Coma followed by death would ensure first before anything else if a
> person has more insulin than s/he needs to become euglycemic.

There can be difference between more and excess. Probably, insulin
resistant people may need more insulin--natural or added and that moy
not be excess to cause hypoglycemia. Insulin insenstiveness may be
meant for it. It it ok?

Can body use Advanced Glycation Endproducts (AGEs) or
/glycated/glycosylated proteins/lipids as a source of glucose/energy?

Can Advanced Glycation Endproducts (AGEs) or /glycated/glycosylated
proteins/lipids trigger inflammatory responses--systemic or local?

Can VAT be glycolipds?

Can ingestion of outside (exogenous) glycated products after absorption
cause excessive inflamatory responses in diabetics?

> May GOD continue to heal your heart by curing your diabetes, dear
> neighbor Kumar whom I love unconditionally.
>
> Prayerfully in Christ's amazing love,
>
> Andrew <><
> --
> Andrew B. Chung
> Cardiologist, Atlanta, Georgia, USA
> http://HeartMDPhD.com/HolySpirit
>
> As for knowing who are the very elect, these you will know by the
> unconditional love they have for everyone including their enemies
> (Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
>
> http://groups.google.com/group/sci.m...ad7fe68478acf?

Kumar wrote:
> Andrew B. Chung, MD/PhD wrote:
> > Kumar wrote:
> > > Andrew B. Chung, MD/PhD wrote:
> > > > Insulin promotes anabolism and not catabolism.
> > >
> > > Yes therefore I mentioned; On other side insulin more exposure may
> > > cause decreased catabolism of these energy stores. Means, more insulin
> > > may resist breakdown of these energy stores?
> >
> > Coma followed by death would ensure first before anything else if a
> > person has more insulin than s/he needs to become euglycemic.
>
> There can be difference between more and excess.

They mean different things.

> Probably, insulin
> resistant people may need more insulin--natural or added and that moy
> not be excess to cause hypoglycemia.

Correct.

> Insulin insenstiveness may be
> meant for it. It it ok?

Ok.

> Can body use Advanced Glycation Endproducts (AGEs) or
> /glycated/glycosylated proteins/lipids as a source of glucose/energy?

Yes, when you are extremely hungry.

> Can Advanced Glycation Endproducts (AGEs) or /glycated/glycosylated
> proteins/lipids trigger inflammatory responses--systemic or local?

Lipids are not glycosylated but they are oxidized.

> Can VAT be glycolipds?

VAT is living pathological tissue.

> Can ingestion of outside (exogenous) glycated products after absorption
> cause excessive inflamatory responses in diabetics?

Not clinically seen.

May GOD continue to heal your heart by curing your dianetes, dear
neighbor Kumar whom I love unconditionally.

Prayerfully in Christ's amazing love,

Andrew <><
--
Andrew B. Chung
Cardiologist, Atlanta, Georgia, USA
http://HeartMDPhD.com/HolySpirit

As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).

http://groups.google.com/group/sci.m...ad7fe68478acf?

Andrew B. Chung, MD/PhD wrote:
> Kumar wrote:
> > Andrew B. Chung, MD/PhD wrote:
> > > Kumar wrote:
> > > > Andrew B. Chung, MD/PhD wrote:
> > > > > Insulin promotes anabolism and not catabolism.
> > > >
> > > > Yes therefore I mentioned; On other side insulin more exposure may
> > > > cause decreased catabolism of these energy stores. Means, more insulin
> > > > may resist breakdown of these energy stores?
> > >
> > > Coma followed by death would ensure first before anything else if a
> > > person has more insulin than s/he needs to become euglycemic.
> >
> > There can be difference between more and excess.
>
> They mean different things.
>
> > Probably, insulin
> > resistant people may need more insulin--natural or added and that may
> > not be excess to cause hypoglycemia.
>
> Correct.
>
> > Insulin insenstiveness may be
> > meant for it. It it ok?
>
> Ok.
>
> > Can body use Advanced Glycation Endproducts (AGEs) or
> > /glycated/glycosylated proteins/lipids as a source of glucose/energy?
>
> Yes, when you are extremely hungry.

Internally, how?

> > Can Advanced Glycation Endproducts (AGEs) or /glycated/glycosylated
> > proteins/lipids trigger inflammatory responses--systemic or local?
>
> Lipids are not glycosylated but they are oxidized.

It is defined as under but not clear much;

"Glycolipids are carbohydrate-attached lipids. Their role is to provide
energy and also serve as markers for cellular recognition.
http://en.wikipedia.org/wiki/Glycolipid "

"Glycation (sometimes called non-enzymatic glycosylation) is the result
of a sugar molecule, such as fructose or glucose, bonding to a protein
or lipid molecule without the controlling action of an enzyme.
Until recently, it was thought that exogenous glycations and AGEs were
negligible contributors to inflammation and disease states, but recent
work has shown that they are important (Vlassara, 2005). Although most
of the research work has been done with reference to diabetes, these
results are most likely important for all people as exogenous AGEs are
implicated in the initiation of retinal dysfunction, cardiovascular
diseases, type II diabetes, and many other age related chronic
diseases.
Endogenous glycations occur mainly in the bloodstream to a small
proportion of the absorbed simple sugars: glucose, fructose and
galactose. The balance of the sugar molecules is used for metabolic
processes. *It appears that fructose and galactose have approximately
ten times the glycation activity of glucose, the primary body fuel
http://en.wikipedia.org/wiki/Glycation "

Anyway, Can exogenous Advanced Glycation Endproducts (AGEs) or
/glycated/glycosylated proteins after absorption trigger
inflammatory(systemic or local) response?

Whether fructose and galactose (fruits intake) are more harmful in view
of above quote?

> > Can VAT be glycolipds?
>
> VAT is living pathological tissue.

Yes, but its stores are fats?

Pls tell me about enegy taken from Triglycerides,Glycerol and FFAs?

> > Can ingestion of outside (exogenous) glycated products after absorption
> > cause excessive inflamatory responses in diabetics?
>
> Not clinically seen.
>
> May GOD continue to heal your heart by curing your dianetes, dear
> neighbor Kumar whom I love unconditionally.
>
> Prayerfully in Christ's amazing love,
>
> Andrew <><
> --
> Andrew B. Chung
> Cardiologist, Atlanta, Georgia, USA
> http://HeartMDPhD.com/HolySpirit
>
> As for knowing who are the very elect, these you will know by the
> unconditional love they have for everyone including their enemies
> (Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
>
> http://groups.google.com/group/sci.m...ad7fe68478acf?

Kumar wrote:
> Andrew B. Chung, MD/PhD wrote:
> > Kumar wrote:
> > > Andrew B. Chung, MD/PhD wrote:
> > > > Kumar wrote:
> > > > > Andrew B. Chung, MD/PhD wrote:
> > > > > > Insulin promotes anabolism and not catabolism.
> > > > >
> > > > > Yes therefore I mentioned; On other side insulin more exposure may
> > > > > cause decreased catabolism of these energy stores. Means, more insulin
> > > > > may resist breakdown of these energy stores?
> > > >
> > > > Coma followed by death would ensure first before anything else if a
> > > > person has more insulin than s/he needs to become euglycemic.
> > >
> > > There can be difference between more and excess.
> >
> > They mean different things.
> >
> > > Probably, insulin
> > > resistant people may need more insulin--natural or added and that may
> > > not be excess to cause hypoglycemia.
> >
> > Correct.
> >
> > > Insulin insenstiveness may be
> > > meant for it. It it ok?
> >
> > Ok.
> >
> > > Can body use Advanced Glycation Endproducts (AGEs) or
> > > /glycated/glycosylated proteins/lipids as a source of glucose/energy?
> >
> > Yes, when you are extremely hungry.
>
> Internally, how?

Hungry monocytes become macrophages that are better able to breakdown
these compounds.

May GOD continue to heal your heart by curing your diabetes, dear
neighbor Kumar whom I love unconditionally.

Prayerfully in Christ's amazing love,

Andrew <><
--
Andrew B. Chung
Cardiologist, Atlanta, Georgia, USA
http://HeartMDPhD.com/HolySpirit

As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).

http://groups.google.com/group/sci.m...ad7fe68478acf?

Andrew B. Chung, MD/PhD wrote:
> Kumar wrote:
> > Andrew B. Chung, MD/PhD wrote:
> > > Kumar wrote:
> > > > Andrew B. Chung, MD/PhD wrote:
> > > > > Kumar wrote:
> > > > > > Andrew B. Chung, MD/PhD wrote:
> > > > > > > Insulin promotes anabolism and not catabolism.
> > > > > >
> > > > > > Yes therefore I mentioned; On other side insulin more exposure may
> > > > > > cause decreased catabolism of these energy stores. Means, more insulin
> > > > > > may resist breakdown of these energy stores?
> > > > >
> > > > > Coma followed by death would ensure first before anything else if a
> > > > > person has more insulin than s/he needs to become euglycemic.
> > > >
> > > > There can be difference between more and excess.
> > >
> > > They mean different things.
> > >
> > > > Probably, insulin
> > > > resistant people may need more insulin--natural or added and that may
> > > > not be excess to cause hypoglycemia.
> > >
> > > Correct.
> > >
> > > > Insulin insenstiveness may be
> > > > meant for it. It it ok?
> > >
> > > Ok.
> > >
> > > > Can body use Advanced Glycation Endproducts (AGEs) or
> > > > /glycated/glycosylated proteins/lipids as a source of glucose/energy?
> > >
> > > Yes, when you are extremely hungry.
> >
> > Internally, how?
>
> Hungry monocytes become macrophages that are better able to breakdown
> these compounds.
Can food with high GI ause excessive glycation/AGEs resuting into
excessive free radicals and inflammatory responses?
> May GOD continue to heal your heart by curing your diabetes, dear
> neighbor Kumar whom I love unconditionally.
>
> Prayerfully in Christ's amazing love,
>
> Andrew <><
> --
> Andrew B. Chung
> Cardiologist, Atlanta, Georgia, USA
> http://HeartMDPhD.com/HolySpirit
>
> As for knowing who are the very elect, these you will know by the
> unconditional love they have for everyone including their enemies
> (Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
>
> http://groups.google.com/group/sci.m...ad7fe68478acf?

Kumar wrote:
> Andrew B. Chung, MD/PhD wrote:
> > Kumar wrote:
> > > Andrew B. Chung, MD/PhD wrote:
> > > > Kumar wrote:
> > > > > Andrew B. Chung, MD/PhD wrote:
> > > > > > Kumar wrote:
> > > > > > > Andrew B. Chung, MD/PhD wrote:
> > > > > > > > Insulin promotes anabolism and not catabolism.
> > > > > > >
> > > > > > > Yes therefore I mentioned; On other side insulin more exposure may
> > > > > > > cause decreased catabolism of these energy stores. Means, more insulin
> > > > > > > may resist breakdown of these energy stores?
> > > > > >
> > > > > > Coma followed by death would ensure first before anything else if a
> > > > > > person has more insulin than s/he needs to become euglycemic.
> > > > >
> > > > > There can be difference between more and excess.
> > > >
> > > > They mean different things.
> > > >
> > > > > Probably, insulin
> > > > > resistant people may need more insulin--natural or added and that may
> > > > > not be excess to cause hypoglycemia.
> > > >
> > > > Correct.
> > > >
> > > > > Insulin insenstiveness may be
> > > > > meant for it. It it ok?
> > > >
> > > > Ok.
> > > >
> > > > > Can body use Advanced Glycation Endproducts (AGEs) or
> > > > > /glycated/glycosylated proteins/lipids as a source of glucose/energy?
> > > >
> > > > Yes, when you are extremely hungry.
> > >
> > > Internally, how?
> >
> > Hungry monocytes become macrophages that are better able to breakdown
> > these compounds.

> Can food with high GI ause excessive glycation/AGEs resuting into
> excessive free radicals and inflammatory responses?

Not in normal folks who don't have visceral adipose tissue (VAT).

May GOD continue to heal your heart by curing your diabetes, dear
neighbor Kumar whom I love unconditionally.

Prayerfully in Christ's amazing love,

Andrew <><
--
Andrew B. Chung
Cardiologist, Atlanta, Georgia, USA
http://HeartMDPhD.com/HolySpirit

As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
http://HeartMDPhD.com/Love

Andrew B. Chung, MD/PhD wrote:
> Kumar wrote:
> > Andrew B. Chung, MD/PhD wrote:
> > > Kumar wrote:
> > > > Andrew B. Chung, MD/PhD wrote:
> > > > > Kumar wrote:
> > > > > > Andrew B. Chung, MD/PhD wrote:
> > > > > > > Kumar wrote:
> > > > > > > > Andrew B. Chung, MD/PhD wrote:
> > > > > > > > > Insulin promotes anabolism and not catabolism.
> > > > > > > >
> > > > > > > > Yes therefore I mentioned; On other side insulin more exposure may
> > > > > > > > cause decreased catabolism of these energy stores. Means, more insulin
> > > > > > > > may resist breakdown of these energy stores?
> > > > > > >
> > > > > > > Coma followed by death would ensure first before anything else if a
> > > > > > > person has more insulin than s/he needs to become euglycemic.
> > > > > >
> > > > > > There can be difference between more and excess.
> > > > >
> > > > > They mean different things.
> > > > >
> > > > > > Probably, insulin
> > > > > > resistant people may need more insulin--natural or added and that may
> > > > > > not be excess to cause hypoglycemia.
> > > > >
> > > > > Correct.
> > > > >
> > > > > > Insulin insenstiveness may be
> > > > > > meant for it. It it ok?
> > > > >
> > > > > Ok.
> > > > >
> > > > > > Can body use Advanced Glycation Endproducts (AGEs) or
> > > > > > /glycated/glycosylated proteins/lipids as a source of glucose/energy?
> > > > >
> > > > > Yes, when you are extremely hungry.
> > > >
> > > > Internally, how?
> > >
> > > Hungry monocytes become macrophages that are better able to breakdown
> > > these compounds.
>
> > Can food with high GI ause excessive glycation/AGEs resuting into
> > excessive free radicals and inflammatory responses?
>
> Not in normal folks who don't have visceral adipose tissue (VAT).

How?
> May GOD continue to heal your heart by curing your diabetes, dear
> neighbor Kumar whom I love unconditionally.
>
> Prayerfully in Christ's amazing love,
>
> Andrew <><
> --
> Andrew B. Chung
> Cardiologist, Atlanta, Georgia, USA
> http://HeartMDPhD.com/HolySpirit
>
> As for knowing who are the very elect, these you will know by the
> unconditional love they have for everyone including their enemies
> (Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
> http://HeartMDPhD.com/Love

"Kumar" <lordshiva5753@rediffmail.com> wrote in message
news:1162314504.823729.196600@m73g2000cwd.googlegr oups.com...
>
> Andrew B. Chung, MD/PhD wrote:
>> Kumar wrote:
>> > Andrew B. Chung, MD/PhD wrote:
>> > > Kumar wrote:
>> > > > Andrew B. Chung, MD/PhD wrote:
>> > > > > Kumar wrote:
>> > > > > > Andrew B. Chung, MD/PhD wrote:
>> > > > > > > Kumar wrote:
>> > > > > > > > Andrew B. Chung, MD/PhD wrote:
>> > > > > > > > > Insulin promotes anabolism and not catabolism.
>> > > > > > > >
>> > > > > > > > Yes therefore I mentioned; On other side insulin more
>> > > > > > > > exposure may
>> > > > > > > > cause decreased catabolism of these energy stores. Means,
>> > > > > > > > more insulin
>> > > > > > > > may resist breakdown of these energy stores?
>> > > > > > >
>> > > > > > > Coma followed by death would ensure first before anything
>> > > > > > > else if a
>> > > > > > > person has more insulin than s/he needs to become euglycemic.
>> > > > > >
>> > > > > > There can be difference between more and excess.
>> > > > >
>> > > > > They mean different things.
>> > > > >
>> > > > > > Probably, insulin
>> > > > > > resistant people may need more insulin--natural or added and
>> > > > > > that may
>> > > > > > not be excess to cause hypoglycemia.
>> > > > >
>> > > > > Correct.
>> > > > >
>> > > > > > Insulin insenstiveness may be
>> > > > > > meant for it. It it ok?
>> > > > >
>> > > > > Ok.
>> > > > >
>> > > > > > Can body use Advanced Glycation Endproducts (AGEs) or
>> > > > > > /glycated/glycosylated proteins/lipids as a source of
>> > > > > > glucose/energy?
>> > > > >
>> > > > > Yes, when you are extremely hungry.
>> > > >
>> > > > Internally, how?
>> > >
>> > > Hungry monocytes become macrophages that are better able to breakdown
>> > > these compounds.
>>
>> > Can food with high GI ause excessive glycation/AGEs resuting into
>> > excessive free radicals and inflammatory responses?
>>
>> Not in normal folks who don't have visceral adipose tissue (VAT).
>
> How?

Why should one teach you while you are arguing?

>> May GOD continue to heal your heart by curing your diabetes, dear
>> neighbor Kumar whom I love unconditionally.
>>
>> Prayerfully in Christ's amazing love,
>>
>> Andrew <><
>> --
>> Andrew B. Chung
>> Cardiologist, Atlanta, Georgia, USA
>> http://HeartMDPhD.com/HolySpirit
>>
>> As for knowing who are the very elect, these you will know by the
>> unconditional love they have for everyone including their enemies
>> (Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
>> http://HeartMDPhD.com/Love
>

The Lord of Fair Punishments wrote:
> "Kumar" <lordshiva5753@rediffmail.com> wrote in message
> news:1162314504.823729.196600@m73g2000cwd.googlegr oups.com...
> >
> > Andrew B. Chung, MD/PhD wrote:
> >> Kumar wrote:
> >> > Andrew B. Chung, MD/PhD wrote:
> >> > > Kumar wrote:
> >> > > > Andrew B. Chung, MD/PhD wrote:
> >> > > > > Kumar wrote:
> >> > > > > > Andrew B. Chung, MD/PhD wrote:
> >> > > > > > > Kumar wrote:
> >> > > > > > > > Andrew B. Chung, MD/PhD wrote:
> >> > > > > > > > > Insulin promotes anabolism and not catabolism.
> >> > > > > > > >
> >> > > > > > > > Yes therefore I mentioned; On other side insulin more
> >> > > > > > > > exposure may
> >> > > > > > > > cause decreased catabolism of these energy stores. Means,
> >> > > > > > > > more insulin
> >> > > > > > > > may resist breakdown of these energy stores?
> >> > > > > > >
> >> > > > > > > Coma followed by death would ensure first before anything
> >> > > > > > > else if a
> >> > > > > > > person has more insulin than s/he needs to become euglycemic.
> >> > > > > >
> >> > > > > > There can be difference between more and excess.
> >> > > > >
> >> > > > > They mean different things.
> >> > > > >
> >> > > > > > Probably, insulin
> >> > > > > > resistant people may need more insulin--natural or added and
> >> > > > > > that may
> >> > > > > > not be excess to cause hypoglycemia.
> >> > > > >
> >> > > > > Correct.
> >> > > > >
> >> > > > > > Insulin insenstiveness may be
> >> > > > > > meant for it. It it ok?
> >> > > > >
> >> > > > > Ok.
> >> > > > >
> >> > > > > > Can body use Advanced Glycation Endproducts (AGEs) or
> >> > > > > > /glycated/glycosylated proteins/lipids as a source of
> >> > > > > > glucose/energy?
> >> > > > >
> >> > > > > Yes, when you are extremely hungry.
> >> > > >
> >> > > > Internally, how?
> >> > >
> >> > > Hungry monocytes become macrophages that are better able to breakdown
> >> > > these compounds.
> >>
> >> > Can food with high GI ause excessive glycation/AGEs resuting into
> >> > excessive free radicals and inflammatory responses?
> >>
> >> Not in normal folks who don't have visceral adipose tissue (VAT).
> >
> > How?
>
> Why should one teach you while you are arguing?

Sorry, you felt like it.

Thanks for much replies. I may now not visit occasionaly for some time
due to some other involvements.

Thanks all again.

Best wishes and greetings.
> >> May GOD continue to heal your heart by curing your diabetes, dear
> >> neighbor Kumar whom I love unconditionally.
> >>
> >> Prayerfully in Christ's amazing love,
> >>
> >> Andrew <><
> >> --
> >> Andrew B. Chung
> >> Cardiologist, Atlanta, Georgia, USA
> >> http://HeartMDPhD.com/HolySpirit
> >>
> >> As for knowing who are the very elect, these you will know by the
> >> unconditional love they have for everyone including their enemies
> >> (Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
> >> http://HeartMDPhD.com/Love
> >

Kumar wrote:
> Andrew B. Chung, MD/PhD wrote:
> > Kumar wrote:
> > > Andrew B. Chung, MD/PhD wrote:
> > > > Kumar wrote:
> > > > > Andrew B. Chung, MD/PhD wrote:
> > > > > > Kumar wrote:
> > > > > > > Andrew B. Chung, MD/PhD wrote:
> > > > > > > > Kumar wrote:
> > > > > > > > > Andrew B. Chung, MD/PhD wrote:
> > > > > > > > > > Insulin promotes anabolism and not catabolism.
> > > > > > > > >
> > > > > > > > > Yes therefore I mentioned; On other side insulin more exposure may
> > > > > > > > > cause decreased catabolism of these energy stores. Means, more insulin
> > > > > > > > > may resist breakdown of these energy stores?
> > > > > > > >
> > > > > > > > Coma followed by death would ensure first before anything else if a
> > > > > > > > person has more insulin than s/he needs to become euglycemic.
> > > > > > >
> > > > > > > There can be difference between more and excess.
> > > > > >
> > > > > > They mean different things.
> > > > > >
> > > > > > > Probably, insulin
> > > > > > > resistant people may need more insulin--natural or added and that may
> > > > > > > not be excess to cause hypoglycemia.
> > > > > >
> > > > > > Correct.
> > > > > >
> > > > > > > Insulin insenstiveness may be
> > > > > > > meant for it. It it ok?
> > > > > >
> > > > > > Ok.
> > > > > >
> > > > > > > Can body use Advanced Glycation Endproducts (AGEs) or
> > > > > > > /glycated/glycosylated proteins/lipids as a source of glucose/energy?
> > > > > >
> > > > > > Yes, when you are extremely hungry.
> > > > >
> > > > > Internally, how?
> > > >
> > > > Hungry monocytes become macrophages that are better able to breakdown
> > > > these compounds.
> >
> > > Can food with high GI ause excessive glycation/AGEs resuting into
> > > excessive free radicals and inflammatory responses?
> >
> > Not in normal folks who don't have visceral adipose tissue (VAT).
>
> How?

http://groups.google.com/group/sci.m...546a7bf4a81ee?

May GOD continue to heal your heart by curing your diabetes, dear
neighbor Kumar whom I love unconditionally.

Prayerfully in Christ's amazing love,

Andrew <><
--
Andrew B. Chung
Cardiologist, Atlanta, Georgia, USA
http://HeartMDPhD.com/HolySpirit

As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
http://HeartMDPhD.com/Love