This article shows that NOT .. 'adhering" .. to .. 'their' ..
advice .. your health .. improves ..
The first article shows CONTRARY to their opinion .. improvement can
and is made and this article shows by going AGAINST .. their advice /
by law approved and going .. "novel" / depletion of iron to what they
consider to be 'near iron deficiency' .. or .. "bad" .. **improves**
health ..
Now in the recent recruiting studies of the effects of iron depletion
in diabetes .. I wonder WHAT the marker for iron they are .. going
to .. use ..?
Near iron deficiency .. ?
I bet .. not ..
<<snip>>
CONCLUSIONS: These results reflect the
insulin-sparing effect of iron
depletion and indicate a key role of iron and hyperinsulinemia in the
pathogenesis of NAFLD
<<snip>>
..
Gastroenterology 2002 Apr;122(4):931-9 Related Articles, Links
Effect of iron depletion in carbohydrate-intolerant patients with
clinical
evidence of nonalcoholic fatty liver disease.
Facchini FS, Hua NW, Stoohs RA.
Department of Medicine, University of California San Francisco and
San
Francisco General Hospital, San Francisco, California, USA.
ffacch...@ecnea.org
BACKGROUND & AIMS: Increased body iron, genetic hemochromatosis (GH)
mutations,
and nonalcoholic fatty liver disease (NAFLD) tend to cluster in
carbohydrate-intolerant patients. In an attempt to further clarify
the
interrelationships among these conditions, we studied 42
carbohydrate-intolerant patients who were free of the common GH
mutations C282Y
and H63D, and had a serum iron saturation lower than 50%. METHODS: We
measured
body iron stores, and induced iron depletion to a level of near-iron
deficiency
(NID) by quantitative phlebotomy. RESULTS: In the 17 patients with
clinical
evidence of NAFLD, we could not demonstrate supranormal levels of
body
iron
(1.6 +/- 0.2 vs. 1.4 +/- 0.2 g; P = 0.06). However, at NID, there was
a
40%-55%
improvement (P = 0.05-0.0001) of both fasting and glucose-stimulated
plasma
insulin concentrations, and near-normalization of serum alanine
aminotransferase activity (from 61 +/- 5 to 32 +/- 2 IU/L; P <
0.001).
CONCLUSIONS: These results reflect the insulin-sparing effect of iron
depletion
and indicate a key role of iron and hyperinsulinemia in the
pathogenesis of
NAFLD.
PMID: 11910345 [PubMed - indexed for MEDLINE]
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Who loves ya.
Tom
Jesus Was A Vegetarian!
http://jesuswasavegetarian.7h.com
Man Is A Herbivore!
http://tinyurl.com/a3cc3
DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk ironjustice@aol.com wrote:
> <<snip>>
> generally supposed to be irreversible
> <<snip>>
>
> Eur J Gastroenterol Hepatol. 2001 Aug;13(8):985-8. Links
> Successful treatment of erectile dysfunction and infertility by
> venesection in a patient with primary haemochromatosis.
> Hamer OW, Gnad M, Scholmerich J, Palitzsch KD.
> Department of Internal Medicine I, University of Regensburg, Germany.
>
> A 36-year-old patient with primary haemochromatosis presented with
> erectile dysfunction. Laboratory findings revealed reduced levels of
> luteinizing hormone (0.4 IU/l; normal range 2-12 IU/l), follicle-
> stimulating hormone (0.1 IU/l; normal range 1-12 IU/l) and
> testosterone (0.49 microg/l; normal range 2-8.1 microg/l). We made the
> diagnosis of secondary hypogonadism due to haemochromatosis, which is
> generally supposed to be irreversible. Due to consequent venesection
> therapy, levels of ferritin and transferrin saturation could be
> normalized, and levels of luteinizing hormone and follicle-stimulating
> hormone increased to normal ranges. Also, testosterone levels became
> normal and remained so without any androgen substitution. The patient
> subsequently regained erectile function and potency. This case
> underlines the fact that a hypogonadotrophic hypogonadism caused by
> iron overload can be reversed by a consequent venesection therapy.
>
> PMID: 11507369 [PubMed - indexed for MEDLINE]
>
> Who loves ya.
> Tom
>
>
> Jesus Was A Vegetarian!
> http://jesuswasavegetarian.7h.com
>
>
> Man Is A Herbivore!
> http://tinyurl.com/a3cc3
>
>
> DEAD PEOPLE WALKING
> http://tinyurl.com/zk9fk